Can in the case of multiple sclerosis (MS),
a too high osmotic pressure in the nerve,
lead to a high intracellular concentration of potassium,
and also lead to 'pumping up' of nerve cells,
which then due to the strongly increased internal pressure,
the insulating myelin sheath can be pressed broken,
resulting in a lesion,
or a strong reduced transmission of stimuli ?
If that were to happen,
one can expect in cerebrospinal fluid,
cytokines, which are normally also a component of the nerve cell contents,
and that it wrongly can be interpreted,
as inflammatory reaction to a potential autoimmune disease.
Is there research of this ?
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